What are the mechanisms of action by which IV acetaminophen exerts pain-relieving properties and how is it different from other agents? Why is this MOA suited for multimodal therapy?

What are the mechanisms of action by which IV acetaminophen exerts pain-relieving properties and how is it different from other agents? Why is this MOA suited for multimodal therapy?

What are the mechanism(s) of action by which IV acetaminophen exerts its pain-relieving properties and how is this differentiated from other agents? Why is this MOA particularly valuable and complementary as part of multimodal therapy in conjunction with opioids?


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Presenter

Jeff Gadsden, MD

Jeff Gadsden, MD

Associate Professor
Duke University School of Medicine
Chief, Division of Orthopaedic, Plastic and Regional Anesthesiology
Duke University Medical Center
Chapel Hill, North Carolina